Cancer & the Black Seed
Scientists at the Cancer Immuno-Biology Laboratory of Hilton Head Island, South Carolina, are looking for a substance that kills cancerous cells without damaging the healthy tissue, as is the case during chemotherapy. The amazing results of their study are explained in the article "Study of Nigella Sativa on Humans," the first scientific report on the cancer-fighting effects of Black Seed Oil. This study shows that Black Seed is superior to other medicines in many regards: Black Seed proved to be more effective than chemotherapy and radiation treatments-and without their serious side effects.
Ideal Substance for Cancer Prevention
The study cites several factors that make Black Seed Oil an ideal substance for cancer prevention. The scientists found that a healthy immune system can recognise tumor cells before they endanger other cells and destroy them. Black Seed Oil helps control tumors in multiple ways:
- It stimulates the production of bone marrow cells.
- It increases the production of immune cells.
- It increases the interferon production.
- Body cells are better protected against damaging influences.
- Tumor cells are destroyed.
- The number of B-cells that produce antibodies is thus increased.
Proven Successes in Cancer Therapy
The Cancer Immuno-Biology Laboratory of South Carolina ran a series of experiments in which mice were infected with tumor cells. Two thirds of the animals treated with Black Seed Oil were still alive thirty days after being infected. In contrast, all of the mice that did not receive Black Seed treatment died in this time period.
Similarly encouraging results come from experiments with human bone marrow cells and tumor cells: a 250 percent increase of bone marrow cells and an almost 50 percent reduction in the growth of tumor cells were directly attributed to the effects of the Black Seed Oil therapy. The ingestion of Black Seed Oil is thus an effective support for every cancer therapy.
Source: Black Cumin the Magical Egyption Herb for Allergies, Asthma, and Immune Disorders.
Peter Schleicher, M.D, and Mohamed Saleh, M.D.
Black Seed Oil has been shown to fight Cancer:
Anti-Angiogenic activity of Nigella Sativa plant extract in cancer therapy.
UNIQUE ID: 98638377
AUTHOR: Medenica R, Janssens J, Tarasenko A, Lazovic G, Corbitt W, Powell D, Jocic D, Mujovic V
ADDRESS: International Immuno-Biology Research Laboratory, South Carolina, 29926
TITLE: Anti-angiogenic activity of nigella sativa plant extract in cancer therapy (Meeting abstract).
SOURCE: Proc Annu Meet Am Assoc Cancer Res; 38:A1377 1997
Nigella sativa [NS], or 'Black Seed', an annual herb belonging to the family Ranunculaceae, has strong immunomodulatory and interferon-like activity. We confirm that our extract of NS inhibits cancer and endothelial cell progression, decreases the production of the angiogenic protein-fibroblastic growth factor (FGF) made by tumor cells, and inhibits growth factor for endothelial cells. FGF-1 and FGF-2 are both localized to myoepithelial and to epithelial cells. FGF is an autocrine growth factor developed in our immortalized human mammary epithelial cells. FGF-2, present in aggressive breast cancer, was suppressed by NS. In addition, our breast cancer cell line contains FGF 1-4. Tumor growth is angiogenesis dependent and neo-vascularization is a crucial determinant of the metastatic potential of the tumor. Formed vessels in tumors are hyper-permeable to plasma protein, due to gaps in the endothelial lining. These vascular abnormalities could facilitate entry of the tumor cells into the circulation. We studied breast cancer, prostate cancer, and melanoma cells for acidic fibroblast growth factor which we isolated and suppressed by NS. We attempted to recapitulate early angiogenic evidence in vitro by developing a model of endothelial growth migration and extracellular matrix interaction. Our in vitro assay revealed that stimulated endothelial cells could produce degradative proteinase and invade the extracellular matrix similarly to tumor cells. Moreover, this model system indicated that a fine-tuned balance between proteinase and proteinase inhibitor regulates vascular morphogenesis and invasion. Migrating endothelial cells produce Type 4 collagenase (member of the matrix metalloproteinase family) and serineproteinase. We demonstrated that specific inhibitors of Type 4 collagenase, general metalloproteinase inhibitors and serineproteinase inhibitors blocked endothelial cell invasion of the extracellular matrix. These inhibitors blocked tumor cell invasion in the same assay. NS was compared with these factors and shown to have the same action. The endothelial cells in culture were reverted to a non-angiogenic state when the angiogenic stimulus is neutralized by NS. The activity of NS blocked the tumor growth and dissemination in metastasis and have remarkable promises for clinical use.
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